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Brain DisordersWithin the last decade, the list of green tea health benefits and therapeutic properties has been growing and expanding in various health-related aspects, starting from preventing cancer and ending with improving our dental health. Recently, one more very important health benefit of this miraculous beverage was discovered by the specialists at the University of Pennsylvania and Boston Biomedical Research Institute (BBRI). American scientists suggest that regular consumption of green tea (to be proper, its component EGCG in combination with DAPH-12 chemical) is linked to lower risks of developing such common brain disorders, as Parkinsons and Alzheimers diseases.

The researches revealed that exposure to the combination of the chemicals DAPH-12 and EGCG can have positive effects and block the development of amyloid plaques in the brains. These plaques are made of proteins and have a property to irritate human brain causing memory loss, loss of motor function and various serious brain disorders, including the fatal ones. This discovery is considered to be an optimistic ending of a long scientific work and experiments directed on finding the chemical which can break up and prevent formation of amyloid plaques in human nervous system and brains.

That is why American scientists are very excited and talk about the importance of the laboratory results received. “These findings are significant because it is the first time a combination of specific chemicals has successfully destroyed diverse forms of amyloids at the same time,” says one of the group leaders, Dr. Martin Duennwald of BBR. However, they underline that further research is necessary to investigate the properties of the chemicals and create a mechanism for possible treatment. “Our findings are certainly preliminary and we need further work to fully comprehend the effects of EGCG in combination with other chemicals on amyloids,” Duennwald says. Those who want to read more about this research can be referred to the most recent issue of Nature Chemical Biology journal.

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